European Psychiatry - Volume 41 - April 2017

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25th European Congress of Psychiatry / European Psychiatry 41S (2017) S106–S169 S157
assessment of both premature mortality (years of life lost–YLLs)
and nonfatal outcomes (years lived with disability–YLDs). DALYs
are computed by adding YLLs and YLDs for each age-sex-
country group. In 2013, mental disorders contributed to 5.6% of
total disease burden in EMR (1894 DALYS/100,000 population):
2519 DALYS/100,000 (2590/100,000 males, 2426/100,000 females)
in high-income countries, 1884 DALYS/100,000 (1618/100,000
males, 2157/100,000 females) in middle-income countries, 1607
DALYS/100,000 (1500/100,000 males, 1717/100,000 females) in
low-income countries. Females had a greater proportion of bur-
den due to mental disorders than did males of equivalent ages,
except for those under 15 years. The highest proportion of DALYs
occurred in the 25–49 age group. The burden of mental dis-
orders in EMR increased from 1726 DALYs/100,000 in 1990 to
1912DALYs/100,000 in 2013 (10.8% increase). Depressive disorders
accounted for most DALYs, followed by anxiety disorders. Pales-
tine had the largest burden of mental disorders. Nearly all EMR
countries had a higher mental disorder burden compared to global
level. Our findings call for EMR health ministries to increase provi-
sion of mental health services and to address stigma of mental ill-
ness. Our results showing the accelerating burden of mental health
are alarming as the region is seeing an increased level of instability.
Disclosure of interest The authors have not supplied their decla-
ration of competing interest.
EW0155
Facial emotion recognition ability in
psychiatrists, psychologist and
psychological counselors
M. Dalkiran ∗, E. Yuksek , O. Karamustafalioglu
1 Sisli Hamidiye Etfal Hospital, Psychiatry, Istanbul, Turkey
2 Viransehir public hospital, Psychiatry, Sanliurfa, Turkey
∗ Corresponding author.
Objectives Although, emotional cues like facial emotion expres-
sions seem to be important in social interaction, there is limited
specific training about emotional cues for psychology professions.
Aims Here, we aimed to evaluate psychologist’, psychological
counselors’ and psychiatrists’ ability of facial emotion recognition
and compare these groups.
Methods One hundred and forty-one master degree students of
clinical psychology and 105 psychiatrists who identified them-
selves as psychopharmacologists were asked to perform facial
emotion recognition test after filling out socio-demographic ques-
tionnaire. The facial emotion recognition test was constructed by
using a set of photographs (happy, sad, fearful, angry, surprised,
disgusted, and neutral faces) from Ekman and Friesen’s.
Results Psychologists were significantly better in recogniz-
ing sad facial emotion than psychopharmacologists (6.23 ±1.08
vs 5.80 ± 1.34 and P = 0.041). Psychological counselors were
significantly better in recognizing sad facial emotion than psy-
chopharmacologists (6.24 ± 1.01 vs 5.80± 1.34 and P = 0.054).
Psychologists were significantly better in recognizing angry facial
emotion than psychopharmacologists (6.54 ± 0.73 vs 6.08 ±1.06
and P = 0.002). Psychological counselors were significantly better
in recognizing angry facial emotion than psychopharmacologists
(6.48 ± 0.73 vs 6.08 ±1.06 and P = 0.14).
Conclusion We have revealed that the pyschologist and psycho-
logical counselors weremore accurate in recognizing sad and angry
facial emotions than psychopharmacologists. We considered that
more accurate recognition of emotional cues may have important
influences on patient doctor relationship. It would be valuable to
investigate how these differences or training the ability of facial
emotion recognition would affect the quality of patient–clinician
interaction.
Keywords Facial emotion recognition; Psychiatrist;
Psychologist; Psychological counselors
Disclosure of interest The authors have not supplied their decla-
ration of competing interest.
EW0156
Family functioning, trauma exposure
and PTSD in a middle-income
community sample
S. Dorrington∗ Zavos , H. Ball , P. McGuffin , A. Sumathipala ,
S. Siribaddana , F. Rijsdijk , S.L. Hatch , M. Hotopf
King’s College London, Institute of Psychiatry, London, United
Kingdom
∗ Corresponding author.
Introduction Only a minority of trauma-exposed individuals go
on to develop post traumatic stress disorder (PTSD). Previous
studies in high-income countries suggest that maladaptive family
functioning adversities (MFFA) in childhood may partially ex-plain
individual variation in vulnerability to PTSD following trauma. We
test in a lower middle income setting (Sri Lanka) whether: (1) MFFA
moderates the association between exposure to trauma and later
(a) PTSD (b) other psychiatric diagnoses; (2) any moderation by
MFFA is explained by experiences of interpersonal violence, cumu-
lative trauma exposure or other psychopathology.
Methods We conducted a population study of 3995 twins and
2019 singletons residing in Colombo, Sri Lanka. Participants com-
pleted the composite international diagnostic interview, including
nine traumatic exposures and a questionnaire on MFFA.
Results In total, 23.4% of participants reported exposure to MFFA.
We found that (1)MFFAmoderates the association between trauma
exposure and both (a) PTSD and (b) non-PTSD diagnosis. (2) This
was not explained by interpersonal violence, cumulative trauma
exposure or other psychopathology.
Conclusions In our sample MFFA moderates the association
between trauma and PTSD, and the association between trauma
and non-PTSD psychopathology.
Disclosure of interest The authors have not supplied their decla-
ration of competing interest.
EW0157
Kbg syndrome and the establishment
of its neuropsychological phenotype
J. Egger ∗, L. Van Dongen , C. Stumpel , E. Wingbermuehle ,
T. Kleefstra
1 Vincent van Gogh Institute, Centre of Excellence for
Neuropsychiatry, Venray, The Netherlands
2 Maastricht University Medical Centre, Department of Genetics,
Maastricht, The Netherlands
3 Radboud University Medical Centre, Department of Genetics,
Nijmegen, The Netherlands
∗ Corresponding author.
Objective KBG syndrome is caused by a mutation in the ANKRD11
gene, characterized by short stature and specific dental, craniofacial
and skeletal anomalies. Scarce literature on the phenotypical pre-
sentation mention delayed speech and motor development as well
asmild tomoderate intellectual disabilities. As to psychopathology,
often, autism and ADHD are mentioned but not yet substantiated
in terms of neurocognitive variables.
Aim Aim of the current study was to investigate neurocognitive
aspects of KBG syndrome.
Participants and Methods Seventeen patients (aged 6–66 years;
ten females) with a proven ANKRD11 mutation were compared
with two different groups of patients with a genetic disorder and
similar developmental ages (n = 14 and n = 10). Neuropsychologi-
cal assessment was performed focusing on the level of intellectual